Science Journal of Public Health

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Homocysteine and Lipid Peroxidation in Active and Passive Smoking

Received: 13 December 2017    Accepted: 12 January 2018    Published: 25 January 2018
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Abstract

Disease risk due to smoking is not limited to smokers only. Passive smoking (exposure to environmental tobacco smoke) is associated with adverse health effect especially in cardiovascular disease. Side-stream cigarette smoke, a major component of secondhand smoke induces reactive oxygen species with promote oxidative stress. This paper summarizes the cardiovascular effects of tobacco smoke. According to the results of the alternative level of biochemistry substances, Cholesterol (C), HDL- Cholesterol (HDL-C), LDL-Cholesterol (LDL-C), Triglyceride (TG), Malondialdehyde (MDA), Conjugate diene (CD), vitamin B12, folate and Homocysteine (Hcy). The results showed that vitamin B12 and Hcy of smokers were significantly higher than those of non-smokers while C, LDL-C, TG and folate were significantly lower than those of non-smokers. More detail of smoking group, the industrial tobacco smokers had HDL-C and vitamin B12 lower than those in non-smokers whereas passive smokers and local handmade tobacco smokers had serum HDL-C and vitamin B12 higher than those in non-smokers. For serum C, LDL-C, TG and folate of all groups of smokers were significantly lower than non-smokers. The industrial tobacco smokers had serum MDA significantly higher than non-smokers but passive smokers and local handmade tobacco smokers had serum MDA lower than non-smokers. The industrial tobacco smokers and passive smokers had serum Hcy significantly higher than non-smokers but local handmade tobacco smokers had serum Hcy lower than non-smokers.

DOI 10.11648/j.sjph.20180602.12
Published in Science Journal of Public Health (Volume 6, Issue 2, March 2018)
Page(s) 43-49
Creative Commons

This is an Open Access article, distributed under the terms of the Creative Commons Attribution 4.0 International License (http://creativecommons.org/licenses/by/4.0/), which permits unrestricted use, distribution and reproduction in any medium or format, provided the original work is properly cited.

Copyright

Copyright © The Author(s), 2024. Published by Science Publishing Group

Keywords

Homocysteine, Lipid Peroxidation, Active and Passive Smoking

References
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Author Information
  • Faculty of Nursing, Siam University, Bangkok, Thailand

  • Faculty of Nursing, Siam University, Bangkok, Thailand

  • Faculty of Nursing, Siam University, Bangkok, Thailand

  • Pramongkutklaw Hospital, Bangkok, Thailand

  • Faculty of Medicine, Ramathibodi Hospital, Mahidol University, Bangkok, Thailand

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    Duangkamol Viroonudomphol, Pornpimon Poomrittikul, Tharntip Jirakanjana, Siriwan Tribanyatkul, Saowanee Kanjanachumpon. (2018). Homocysteine and Lipid Peroxidation in Active and Passive Smoking. Science Journal of Public Health, 6(2), 43-49. https://doi.org/10.11648/j.sjph.20180602.12

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    ACS Style

    Duangkamol Viroonudomphol; Pornpimon Poomrittikul; Tharntip Jirakanjana; Siriwan Tribanyatkul; Saowanee Kanjanachumpon. Homocysteine and Lipid Peroxidation in Active and Passive Smoking. Sci. J. Public Health 2018, 6(2), 43-49. doi: 10.11648/j.sjph.20180602.12

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    AMA Style

    Duangkamol Viroonudomphol, Pornpimon Poomrittikul, Tharntip Jirakanjana, Siriwan Tribanyatkul, Saowanee Kanjanachumpon. Homocysteine and Lipid Peroxidation in Active and Passive Smoking. Sci J Public Health. 2018;6(2):43-49. doi: 10.11648/j.sjph.20180602.12

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  • @article{10.11648/j.sjph.20180602.12,
      author = {Duangkamol Viroonudomphol and Pornpimon Poomrittikul and Tharntip Jirakanjana and Siriwan Tribanyatkul and Saowanee Kanjanachumpon},
      title = {Homocysteine and Lipid Peroxidation in Active and Passive Smoking},
      journal = {Science Journal of Public Health},
      volume = {6},
      number = {2},
      pages = {43-49},
      doi = {10.11648/j.sjph.20180602.12},
      url = {https://doi.org/10.11648/j.sjph.20180602.12},
      eprint = {https://download.sciencepg.com/pdf/10.11648.j.sjph.20180602.12},
      abstract = {Disease risk due to smoking is not limited to smokers only. Passive smoking (exposure to environmental tobacco smoke) is associated with adverse health effect especially in cardiovascular disease. Side-stream cigarette smoke, a major component of secondhand smoke induces reactive oxygen species with promote oxidative stress. This paper summarizes the cardiovascular effects of tobacco smoke. According to the results of the alternative level of biochemistry substances, Cholesterol (C), HDL- Cholesterol (HDL-C), LDL-Cholesterol (LDL-C), Triglyceride (TG), Malondialdehyde (MDA), Conjugate diene (CD), vitamin B12, folate and Homocysteine (Hcy). The results showed that vitamin B12 and Hcy of smokers were significantly higher than those of non-smokers while C, LDL-C, TG and folate were significantly lower than those of non-smokers. More detail of smoking group, the industrial tobacco smokers had HDL-C and vitamin B12 lower than those in non-smokers whereas passive smokers and local handmade tobacco smokers had serum HDL-C and vitamin B12 higher than those in non-smokers. For serum C, LDL-C, TG and folate of all groups of smokers were significantly lower than non-smokers. The industrial tobacco smokers had serum MDA significantly higher than non-smokers but passive smokers and local handmade tobacco smokers had serum MDA lower than non-smokers. The industrial tobacco smokers and passive smokers had serum Hcy significantly higher than non-smokers but local handmade tobacco smokers had serum Hcy lower than non-smokers.},
     year = {2018}
    }
    

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  • TY  - JOUR
    T1  - Homocysteine and Lipid Peroxidation in Active and Passive Smoking
    AU  - Duangkamol Viroonudomphol
    AU  - Pornpimon Poomrittikul
    AU  - Tharntip Jirakanjana
    AU  - Siriwan Tribanyatkul
    AU  - Saowanee Kanjanachumpon
    Y1  - 2018/01/25
    PY  - 2018
    N1  - https://doi.org/10.11648/j.sjph.20180602.12
    DO  - 10.11648/j.sjph.20180602.12
    T2  - Science Journal of Public Health
    JF  - Science Journal of Public Health
    JO  - Science Journal of Public Health
    SP  - 43
    EP  - 49
    PB  - Science Publishing Group
    SN  - 2328-7950
    UR  - https://doi.org/10.11648/j.sjph.20180602.12
    AB  - Disease risk due to smoking is not limited to smokers only. Passive smoking (exposure to environmental tobacco smoke) is associated with adverse health effect especially in cardiovascular disease. Side-stream cigarette smoke, a major component of secondhand smoke induces reactive oxygen species with promote oxidative stress. This paper summarizes the cardiovascular effects of tobacco smoke. According to the results of the alternative level of biochemistry substances, Cholesterol (C), HDL- Cholesterol (HDL-C), LDL-Cholesterol (LDL-C), Triglyceride (TG), Malondialdehyde (MDA), Conjugate diene (CD), vitamin B12, folate and Homocysteine (Hcy). The results showed that vitamin B12 and Hcy of smokers were significantly higher than those of non-smokers while C, LDL-C, TG and folate were significantly lower than those of non-smokers. More detail of smoking group, the industrial tobacco smokers had HDL-C and vitamin B12 lower than those in non-smokers whereas passive smokers and local handmade tobacco smokers had serum HDL-C and vitamin B12 higher than those in non-smokers. For serum C, LDL-C, TG and folate of all groups of smokers were significantly lower than non-smokers. The industrial tobacco smokers had serum MDA significantly higher than non-smokers but passive smokers and local handmade tobacco smokers had serum MDA lower than non-smokers. The industrial tobacco smokers and passive smokers had serum Hcy significantly higher than non-smokers but local handmade tobacco smokers had serum Hcy lower than non-smokers.
    VL  - 6
    IS  - 2
    ER  - 

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